Why Fats Don’t Make Patients Fat


Research shows that glycemic carbohydrates make patients fat. Parent essential oils (PEOs) are much too precious to cell structure and eicosanoid production to be merely “burned for energy”. The Eicosanoids are compounds that influence a network of controls in the body, particularly immunity and inflammation. The Basic Medical Biochemistry – A Clinical Approach, one of the most renowned medical textbooks on the subject tells us on page 510:

“Adipose tissue [body fat] lacks glycerol kinase and can produce glycerol-3 phosphate only from glucose dihydroxyacetone phosphate [from eating carbohydrates]. Thus, adipose tissue can store fatty acids only when glycolysis is activated, i.e., the fed state [after eating.”

The body fat lacks glycerol kinase, therefore the glycerol-3 phosphate from eating dietary carbohydrate is required. You can’t get it any other way. Page 790 of the textbook gives further insight into patients staying lean-for-life:

“If glycerol-3 phosphate is abundant [from carbohydrates], many of the fatty acids so formed are re-esterified [converted back to triacylglycerols [more body fats]…”

Here’s more proof, from student Companion to Satryr’s Biochemistry, page 610:

“Adipose cells [body fat] constantly break down and resynthesize triacylglycerols, but synthesis [of more body fat] cannot proceed without an external supply of glucose. Thus, externally supplied glucose [from food] is required”.

Therefore, eating fat cannot make patients fat. However, excess consumption of non PEOs, saturated and mono-saturated fats inhibits the burning of excess body fat for energy. The medical textbooks are quite clear about how patients get fat as well as how they can stay lean-for-life.


Mike Maunu – Founder