Why PEOs Are The Solution To EFA Deficiency

EFA DeficiencyD.F. Horrobin, MD, PhD,discusses the lack of a sufficient definition of “normal.”

“… Normal“ [EFA levels] in this context means ‘usual for the population’ and also ‘not obviously diseased.’ It does not necessarily mean that ‘normal’ levels are ones which are optimal for long-term health. Any ‘normal’ Western population has large numbers of people within it who will suffer relatively prematurely from heart disease, cancer, arthritis, dementia and a whole range of other conditions. Estimates of ‘optimal’ EFA levels will only come as the result of large scale prospective studies in which EFA analyses are performed on blood samples from large numbers of apparently healthy individuals, and these people are monitored over decades for the emergence of health problems.” [Note: This HASN’T been done.]

Just as the LDL-C “cholesterol number” has been modified downward with no scientific basis. The FAILURE of fish oil’s EPA / DHA to positively influence Alzheimer’s in patients with low DHA levels. This is direct proof of Dr. Horrobin’s assertion.

Dr. Horrobin covers the critical relationship of PEOs to proteins: “The lipid configuration of the membrane is important in itself, but also matters because it influences the structure and behavior of the many proteins in the membrane such as ion channels, receptors and ATPases [including insulin receptivity]. These proteins are literally afloat in a lipid sea [PEOs] and their function is dependent on the behaviour of that sea.”

The brain and nervous system has the greatest density of omega 3 derivatives—about 14% of its total lipid is EPA / DHA. However, arachidonic acid (AA), an omega 6 derivative, accounts for a significant 10% of total lipid content. Fish oil advocates don’t mention this. Furthermore, adding DHA does nothing to improve dementia. This is a red flag that must be heeded by those who think more supplemental fish oil and DHA is required.


Mike Maunu – Founder